ransferase; CYP735A: cytokinin trans-hydroxylase; CKX: cytokinin dehydrogenase; NCED: 9-cis-epoxycarotenoid dioxygenase; ABA8ox: (+)-abscisic acid 8'-hydroxylase; GPS:

ransferase; CYP735A: cytokinin trans-hydroxylase; CKX: cytokinin dehydrogenase; NCED: 9-cis-epoxycarotenoid dioxygenase; ABA8ox: (+)-abscisic acid 8′-hydroxylase; GPS: ent-copalyl diphosphate synthase; GA3ox: gibberellin three beta-dioxygenase; KS: ent-kaurene synthase; KAO: entkaurenoic acid monooxygenase; ACO: 1-aminocyclopropane-1-carboxylate oxidase; AUX/IAA: auxin-responsive protein IAA; GH3: auxin responsive GH3 gene loved ones; SAUR: SAUR family members proteins; AUX1: auxin influx carrier 1; CRE1: cytokinin receptor 1; B-ARR: two-component response regulator ARR-B household; A-ARR: two-component response regulator ARR-A loved ones; SnRK2: serine/threonine-protein kinase SRK2; ABF: ABA responsive element binding issue; PYL: abscisic acid receptor PYR/PYL household; PP2C: serine/threonineprotein phosphatase 2A catalytic subunit; BSK: BR-signaling kinase; TCH4: xyloglucan:xyloglucosyl transferase TCH4; JAZ: jasmonate ZIM domaincontaining protein; COI-1: coronatine-insensitive protein 1; PR1: standard salivary proline-rich protein 1; NPR1: nonexpresser of pathogenesis-related gene 1; GID1: gibberellin receptor GID1; GID2: F-box protein GID2.Funding Our operate were funded by the Organic CDK6 web Science Foundation of China (No. 31770613) and Opening Project of Zhejiang Provincial Preponderant and Characteristic Subject of Key University (Traditional Chinese Pharmacology), Zhejiang Chinese Health-related University (No. ZYAOXYB2019009 and No. ZYAOX2018004). Availability of data and materials The datasets generated and analysed through the current study are obtainable in the NCBI Short Study Archive with accession quantity PRJNA751266.DeclarationsEthics approval and consent to participate Not applicable. Consent for publication Not applicable. Competing interests The authors declare that they have no competing interests. Author specifics 1 College of Life Science, Jiangsu Standard University, Xuzhou, Jiangsu 221116, People’s Republic of China. two Laboratory of Medicinal Plant Biotechnology, College of Pharmacy, Zhejiang Chinese Health-related University, Hangzhou, Zhejiang 310053, People’s Republic of China. Received: 17 August 2021 Accepted: four LPAR1 MedChemExpress NovemberSupplementary InformationThe on the net version contains supplementary material available at doi. org/10.1186/s12870-021-03396-6. More file 1: Table S1. Primes of chosen target genes for qRT-PCR. Extra file 2: Table S2. The detail data of raw reads from different sample groups. (XLS 21 kb) Further file three: Figure S1. Principal elements evaluation with the four transcriptomes. Further file four. Assembled unigenes within this study. More file five: Figure S2. GO and KEGG annotation and KOG category classification of all unigenes. Extra file six: Figure S3. GO classification of DEGs at 0.5 h (a) and 6 h immediately after KL27-FB remedy (b). Further file 7: Table S3. GO and KEGG enrichment analysis. (XLS 20 kb) Extra file 8: Table S4. DEGs involving in KEGG pathways following KL27FB therapy. (XLS 202 kb) Additional file 9: Table S5. Differential expression of all unigenes in phenylpropanoid biosynthesis pathway (ko00940). (XLS 81 kb) More file 10: Table S6. Differential expression of random chosen genes. More file 11: Table S7. Annotation of unigenes. (XLS 8884 kb) More file 12: Figure S4. Hormone metabolism and signal transduction of auxin (a), CTY (b), ABA (c), ET (d), BR (e), JA (f ), SA (g) and GA (h) following KL27-FB treatment. Extra file 13: Table S7. Annotation to encode putative TFs. (XLS 943 kb) Additional f